13 results
Inflammatory, Immunologic, and Pathobiologic Features Leading to Severe Asthma. #Pathophysiology #Asthma #Mechanism #Leukotriene #Type2 #Inflammatory #Immunologic
Asthma #Mechanism #Leukotriene
Pharmacology: Anti-Allergic Therapy

#Pharmacology #Mechanisms #Allergy #Antiallergic #AntiAllergen #Antihistamines #Leukotrienes
Antihistamines #Leukotrienes
Arachidonic acid pathway and drugs which affect it.

#Pharmacology #Pathophysiology #ArachidonicAcid #Pathways #COX #Prostaglandins #Prostacyclins #Leukotrienes #Synthesis

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Prostacyclins #Leukotrienes
Summary of Treatments for Allergic Rhinitis
 - Intranasal corticosteroids
 - Oral antihistamines
 - Combination intranasal corticosteroid
anticholinergics - Leukotriene
Role of Uric Acid in the Inflammation of Gout

Allopurinol, Febuxostat - Xanthine oxidase
Colchicine - Release of
Colchicine - Release of leukotrienes
Adverse Effects of NSAIDs
Prostaglandins decreased
 - Gastric mucosal damage with ulcer formation, bleeding, and perforation
 -
uterine motility Leukotrienes
The 2019 Global Initiative for Asthma (GINA) treatment strategy figure for adults and adolescents, annotated to
agonists; LTRA: leukotriene
Stepwise Treatment of Chronic Urticaria
Step 1 - Start second-generation HI antihistamine 

Step 2 - One or
night - Add leukotriene
Anaphylaxis: Pathogenesis
Anaphylaxis is a Type 1 hypersensitivity reaction
While the primary mechanism of anaphylaxis is mast cell
prostaglandins, leukotrienes
Pharmacologic options for the treatment of allergic rhinitis include intranasal corticosteroids, oral and intranasal antihistamines, decongestants,
anticholinergics, and leukotriene