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diagnosis pathophysiology causes differential endocrinology nephrology pediatrics pharmacology table actions adrenergic adrenoceptor comparison diarrhea fatal glucagon gluconeogenesis hepatic hepatology hypothyroid
Depostion of glycosamineglycans (mucin) Filtration of albumin Nat retention + < Edema Effusion, hoarse voice, puffed-up face Impaired cerebral development Myxedema Neuromuscular excitability Sensory disturbances Hyporeflexia Lack of drive Depression Clouded
Elimination of drugs #Glycogenolysis
Adrenoceptor Actions α1 • Vascular smooth muscle - Contraction • Genitourinary smooth muscle - Contraction • Intestinal
excitability • Liver - Gycogenolysis ... Relaxation • Liver - Glycogenolysis ... Skeletal muscle - Glycogenolysis
Insulin and Glucagon Pathophysiology Insulin: • Glucose utilization (Glycolysis) • Glucose storage (Glycogenesis) • Fat synthesis
Glycogen breakdown (Glycogenolysis
Hypothyroid Myopathy Very common (up to 80% ) in patients with hypothyroidism Pathophysiology: • T4 deficiency leads to
leads to abnormal glycogenotysis
Commonly Observed Laboratory Findings in Hepatic / Liver Failure - Elevated conjugated and unconjugated bilirubin -
fasting state after glycogenolysis
One Pill Kills - Those that can be fatal even after just ingesting a small amount 1)
to blockage of glycogenolysis
Stimulation of glycogenolysis, gluconeogenesis Hyperglycemia Accelerated breakdown of VLDL and LDL, stimulation of bile secretion VLDL + Cholesterol Energy metabolism† 02 con- sumption Heat metabolism
Stimulation of glycogenolysis
Rhabdomyolysis - Differential Diagnosis Framework Rhabdomyolysis = Destruction of skeletal muscle with leakage of contents into circulation.
Defects: Glycolysis, Glycogenolysis
Metabolic Causes of Kidney Stones in Children • Calcium Oxalate: Primary Hyperoxaluria type 1-3, Secondary Hyperoxaluria
Lesch-Nyhan syndrome, Glycogenosis
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