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pathophysiology pharmacology dyslipidemia primarycare 2013 2017 @dharsaty abnormal accaha acid algorithm amenorrhea atherosclerosis bile bileacids biliary bilirubun cardiology causes cholesterol
Mechanism of Action of Bile Acid Sequestrants Bile Acid Sequestrants → ↓ Hepatic Bile Acid Pool →
Expression → ↑ VLDL ... /Secretion → ↑ LDL ... Production → ↑ LDL ... Receptors → ↑ LDL ... #pharmacology #bileacid
Lipoprotein Metabolism Enterocytes release absorbed lipids the form of triglyceride-rich chylomicrons. These are acted upon by endothelial
lipoproteins (VLDLs ... VLDLs are released ... liproprotein (LDL
ACC/AHA Statin Guidelines - 2013 vs 2017 In 2013 the ACC/AHA guidelines drastically changed the way we
the traditional LDL ... group, if the LDL ... alternatively use the LDL ... of the target LDL ... expect a >50% LDL
Prescription Fish Oils Prescription fish oils, Lovaza, and Vascepa, are commonly used for treatment of hypertriglyceridemia (TG
of increasing LDL ... without effect on LDL
Risk factors for atherosclerosis Modifiable risk factors: - Dyslipidemia (elevated LDL, decreased HDL) - Tobacco smoking -
Dyslipidemia (elevated LDL
Abnormal Lipid Profiles - Differential Diagnosis Algorithm Increased Cholesterol and Triglycerides - Genetic Causes
Androgens Increased LDL
#Estrogen 25-OH-D3 formation #HDL #LDL #VLDL Y Proliferation and desquamation of #vaginalepithelium In children: delayed epiphyseal fusion #Osteoporosis Atherosclerosis Vaginal infections Tall stature #Progestogen #mammarygland tubes No buildup of uterus
formation #HDL #LDL ... #VLDL Y Proliferation
Fibrates - Gemfibrozil and Fenofibrate Mechanism: activates PPAR alpha Lipid Effects: - Decrease TG by 30-50% -
- Decrease LDL
Bile acids are the end products of cholesterol catabolism. Cholic acid and chenodeoxycholic acid are the
diabetes, #bile, #bileacids
Stimulation of glycogenolysis, gluconeogenesis Hyperglycemia Accelerated breakdown of VLDL and LDL, stimulation of bile secretion VLDL + Cholesterol Energy metabolism† 02 con- sumption Heat metabolism
breakdown of VLDL ... and LDL, stimulation ... bile secretion VLDL
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