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diagnosis atlas clinical differential pocus echocardiogram cardiology morphology key microscopy pathology causes pathophysiology algorithm comparison dermatology fungal nephrology nomenclature urine
Digoxin Pharmacology and Pathophysiology 1) Na/K ATPase Pump - Digoxin works by directly binding to and inhibiting
Pathophysiology 1) Na/K ATPase ... of the Na+/K+ ATPase ... blocks the Na+/K+ ATPase ... of the Na+/K+ ATPase ... to the Na+/K+ ATPase
Causes of Hypokalemia - Differential Diagnosis Algorithm Increased Loss - Increased GI Loss - Increased Urinary
Stimulation of Na/K-ATPase
#1,25-(OH)-D Ca-+ Nat ER Na Cytoplasmic receptor Nuclear receptor DNA mRNA #Lipid synthesis Golgi body Protein synthesis Membrane lipids CaBP #ATPase #Ca-+ Blood #ATPase #Basal membrane #Mitochondrion Vesicle? #Cytoplasmic #CaBP (Ca-binding protein) Brush border Ca?+
lipids CaBP #ATPase ... #Ca-+ Blood #ATPase
Drugs that Impair Renal Potassium Excretion Decreased ENaC channel function: Amiloride, Triamterene, Pentamidine Decreased Aldosterone Synthesis: Heparin,
Decreased Na, K-ATPase
Digoxin effect on Electrocardiogram (EKG) Digoxin is a cardiac glycoside from the foxglove leaf Mechanism - Inhibits Na-K
Inhibits Na-K ATPase
Inherited Defects in Kidney Tubule Epithelial Cells Renal glucosuria - Na+-dependent glucose cotransporter Cystinuria - Amino acid transporter Bartter
- exchanger,H+-ATPase
The Effect of Zinc on the Enteral Absorption of Copper. Panel A shows how copper (Cu)
a transporting ATPase
Distal Renal Tubular Acidosis Distal RTA is the true Nephrogenic RTA and can be truly divided into
the luminal H ATPase
The chemical formula of Digoxin is C41 H64 O14. Digoxin (Cardiac glycoside) reversibly inhibits the sodium-potassium-ATPase, causing
sodium-potassium-ATPase ... sodium-potassium-ATPase
Lumen Luminal - membrane Nat-K+ ATPase 1 Primary active Nat and K+ transport Blood Na Nat 145 mmol/L Basolateral membrane Na 15 mmol/L 2 4[Na*] = Chemical driving force for Nat Nat -70 m/ -70
membrane Nat-K+ ATPase
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